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生物通
11月
揭秘结核杆菌 “逃逸术”:ESAT-6 如何借纤维化破坏吞噬体助力感染?
为解决Mycobacterium tuberculosis(Mtb)的 ESAT-6 介导吞噬体膜破坏机制不明的问题,印度国家科学教育与研究学院等机构的研究人员展开研究,发现 ESAT-6 经聚合作用使吞噬体样小室重塑和囊泡化。该成果为理解 Mtb 致病机制及开发新疗法提供依据,值得科研人员一读。
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Trespassing charge dropped
Subpoenas Walz and others
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