研究结论表明，突尼斯人群IBC具有复杂且异质性的遗传背景，涉及DNA修复缺陷和炎症通路异常的双重机制。这种遗传多样性可能反映了突尼斯人群的混合祖先背景（柏柏尔人、阿拉伯人、安达卢西亚人等）。值得注意的是，大多数患者携带多个中高风险变异，支持了IBC遗传易感性的多基因模型。

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